My experience has been that the docs we’ve seen are not really looking for external communicating hydrocephalus. My understanding is that ventricle enlargement would not necessarily occur in external communicating hydro since there is not an obstruction between the ventricles. My sense is that most docs looking at hydro stop once they see no evidence of ventricle enlargement. This is unfortunate since it could be adding to the number of undiagnosed patients with this health problem.
Where did you acquire your understanding from please ? Is it documented anywhere ?
Any ideas how can we change this, so the radiologists start to recognise it ?
Regards
Barbara
(UK)
Head & Neck Injury (June 2002); Mild Concussion; Post Concussion Syndrome; Postural Orthostatic Tachycardia Syndrome (POTS); Peripheral Vestibular Dysfunction; Mild Radiculopathy & Small Fibre Neuropathy (right leg & foot resp.); Partially Empty Sella (Oct 2002) Fully Blown Empty Sella (Oct 2004); Whiplash Associated Disorder (WAD); Cerebellar Ectopia (Chiari 0); Cranio-cervical Instability (CCI) with Posterior Gliding (PG) & Cranial Settling (CS); Brain Compression; Retroflexed Odontoid; Stretched/Elongated Brainstem; Vitamin D deficiency; Ehlers Danlos (EDS) type 111; and now Osteoarthritis!